I was interesting, how animals in animal model were made diabetics?
It is really very interesting point, how animals got diabetes in first place? In all lab any medicine first tested on animals, rat or mice, cats and gods, rabbits and pigs, and so on. Because of obesity is cause of diabetes type 2, as it is stated by ADA animals must be developed to became obese. Is this really true for human diabetes type 2 development? Not at all. Is is not real way we live. So there must be another way to diabet animals, right?
This way is surgical. In animal part of pancreas cut off and another part is left. Animal became diabetic. So, them bigger part of pancreas removed then severe diabetes and faster animal will gone. Regardless of any body size, or anything else, diabetic animal life speed away according to the portion of removed pancreas.
Now, according to ADA we developed diabetes because of our obesity. Still, the test to confirm diabetes based on the level of sugar in blood and there is no one test to check up the health of pancreas. At least no one test ever was given to me. So, I assume, not too many other diabetics type 2 ever had this test. Difference with animals, our pancreas still in the same size as we were born. But probably even bigger due to edema. There is no one test to confirm my b-cells in right healthy amount, and well working condition. No any test to confirm the health and condition of b-cells and pancreas. So, if animals develop diabetes after partial pancreas removal, is this possible that obesity play right opposite role as it is presented by ADA? What if obesity help us to survive? It is simple very logical to assume that pancreas diabetics type 2 decline in health with time and age.
Another very logical thinking that diabetes type 1 is no one way to absolute absence of insulin secretion. If this was the case then diabetics type 1 needed about the same dose of insulin, and never this dose would go down. The same as in animals whose pancreas was removed in full. These animals never recovered from diabetes. They did not have pancreas, so insulin secretion was not possible. It is absolutely different with human. Human diabetics type 1 take very small dose of insulin, the same type of insulin as diabetics type 2. There is no insulin for type of diabetes, such as insulin for type 2 diabetics or for type 1 diabetics. We take the same type of insulin regardless of type of diabetes in our medical charts. With time, diabetics type 1 dose of insulin decreasing.
The severity of diabetes and dose of insulin needed to support life of diabetic animal depend on the portion of pancreas which was removed, not from the size of the body. Then bigger cut then shorter life of diabetic animal.
So, the question is, how studies support the idea that obesity is to blame we are diabetics type 2? What if the pancreas in our bodies is ill, does not secret insulin we all, humans and animals, need to live? In this case then bigger portion of destroyed pancreas in human then severely diabetes type 2, and so the dose of insulin would be higher.
It is really very interesting point, how animals got diabetes in first place? In all lab any medicine first tested on animals, rat or mice, cats and gods, rabbits and pigs, and so on. Because of obesity is cause of diabetes type 2, as it is stated by ADA animals must be developed to became obese. Is this really true for human diabetes type 2 development? Not at all. Is is not real way we live. So there must be another way to diabet animals, right?
This way is surgical. In animal part of pancreas cut off and another part is left. Animal became diabetic. So, them bigger part of pancreas removed then severe diabetes and faster animal will gone. Regardless of any body size, or anything else, diabetic animal life speed away according to the portion of removed pancreas.
Now, according to ADA we developed diabetes because of our obesity. Still, the test to confirm diabetes based on the level of sugar in blood and there is no one test to check up the health of pancreas. At least no one test ever was given to me. So, I assume, not too many other diabetics type 2 ever had this test. Difference with animals, our pancreas still in the same size as we were born. But probably even bigger due to edema. There is no one test to confirm my b-cells in right healthy amount, and well working condition. No any test to confirm the health and condition of b-cells and pancreas. So, if animals develop diabetes after partial pancreas removal, is this possible that obesity play right opposite role as it is presented by ADA? What if obesity help us to survive? It is simple very logical to assume that pancreas diabetics type 2 decline in health with time and age.
Another very logical thinking that diabetes type 1 is no one way to absolute absence of insulin secretion. If this was the case then diabetics type 1 needed about the same dose of insulin, and never this dose would go down. The same as in animals whose pancreas was removed in full. These animals never recovered from diabetes. They did not have pancreas, so insulin secretion was not possible. It is absolutely different with human. Human diabetics type 1 take very small dose of insulin, the same type of insulin as diabetics type 2. There is no insulin for type of diabetes, such as insulin for type 2 diabetics or for type 1 diabetics. We take the same type of insulin regardless of type of diabetes in our medical charts. With time, diabetics type 1 dose of insulin decreasing.
The severity of diabetes and dose of insulin needed to support life of diabetic animal depend on the portion of pancreas which was removed, not from the size of the body. Then bigger cut then shorter life of diabetic animal.
So, the question is, how studies support the idea that obesity is to blame we are diabetics type 2? What if the pancreas in our bodies is ill, does not secret insulin we all, humans and animals, need to live? In this case then bigger portion of destroyed pancreas in human then severely diabetes type 2, and so the dose of insulin would be higher.
via Ravenvoron
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