Diabetes: Immune system can regulate insulin, so what? November 25, 2017

Inflammation processes are responsible for the failure of insulin production in diabetes patients. The patients' own immune systems can contribute to treatment of this disease: researchers have found a feedback mechanism that could help maintain insulin production in overweight sufferers.
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How it is easy. Just read it. The publication is small, easy to read, and open all the absurd we will find in every one of presentations and studies like that.

 In their study, the Basel-based researchers focused specifically on recently discovered ILC2 immune cells in the pancreas, where, under diabetic conditions, the protein IL33 is activated, among others.

What is "Diabetic Condition"? No one clue. So under this junk studies it can be studied all what one wish and presented any result studier wish to publish. Shame!

 This protein stimulates the ILC2 cells, which trigger the release of insulin in overweight individuals using retinoic acid and could therefore be used to inhibit the failure of insulin production.

Another scam. ILC2 stimulated in overweight people, right? What about underweight people, does ILC2 stimulated in them? Those overweight are adults or children? What is the cause of diabetes they do have? As long as I do remember there are many different causes of diabetes. In one case it is fibrosis, in another case it is hyaline changes. And it can be both at the same time in the same individual. Do not mention other pathology of diabetes. So, in what case Ilc2 stimulated to release insulin, and when this stimulation is failed?
BTW, if it was study of individuals under diabetic condition, then probably it is not head but it is tail, the result of diabetes condition rather then normal non diabetic insulin release, right?

It is already known that obesity and diabetes lead to an excessive, pathological activation of the immune system in which the messenger substance IL1-beta plays a central role. This results in the death of insulin-producing cells.

True? false. If this was true then there would not be overweight people without diabetes. Operatic Sopranos really overweight. They could not sing if they diabetics. Diabetic's lungs are severely effected by medical condition we do have. This is why we do have asthma and COPD.
    What if true is on the other end of the story? What if immune system activated because of diabetes,  fibrosis, other beta cells destruction? We all do know that diabetics type 2 can live about ten years after being diagnosed with diabetes type 2. No more. If diabetic type 2 does not start to take insulin early then less then ten years and diabetic will gone: stroke or heart attack, high sugar or low sugar, appendices or flu, Asthma or pneumonia. 

However, if IL1-beta is blocked, diabetes and its complications -- in particular cardiovascular diseases -- can be inhibited. Diabetic inflammatory reactions are already finding use in clinical applications.

This is why I try to avoid hospital as much as I can. We are just Guinea Human Rats for them, MD and Labs.  I hate to be Rat. I try to find MD who will see me as person with all my pain and surviving energy.
    Really what this studies about? What they found? What did they were looking at? That's right, $$$$$$, and they found it. Simple. Brilliant. Shameless.

via Ravenvoron