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Thursday, 3 November 2016

What Caused Insulin Resistance? November 3, 2016

Why people with diabetes prone to heart attacks and strokes? Now researchers at Washington University School of Medicine in St. Louis have found why diabetics have heart attacks and strokes, and die as a result of that. How did they find it? Very simple.
Too much fat in the diet promotes insulin resistance by spurring chronic inflammation. In the image above, immune cells (shown in green) produce fatty acids that contribute to diabetes-related inflammation. Researchers at Washington University School of Medicine have developed a way to block production of fatty acids in these immune cells in mice and protect them from diet-induced diabetes.
      Of cause I did not copy image, do not see any reason for that. All the studies just scam, usual medical scam Organized Medicine feed the public.
       At first, it is taken for granted that high fat diet promoted insulin resistance in mice. What does it means? Medical Industry still not able to find the answer "Insulin Resistance, what is it"? Still they do know for sure, it is result of high fat diet. So, they fed mice with high fat diet, did not let mice to be active so mice became obese and then they stopped to kill the mice and all the problems  of mice gone. Now they present they can do it with humans in the same way. So simple. Let me say it, in first was mice born with diabetes? Not, it was not diabetic before it was in cage ready for studies. Now, how forcefully induced diet changed mice's immune system?
Ok, let me put it in another way, to be more clear. One has very unhealthy lifestyle resulted natural disaster, catastrophe,  war, accident, illness of any kind. There were no one way to live healthy if there is no clean water, no food at all, no medicine or warm clothing. The result of stress, human became diabetic. Now what? Someone would recovery after accident resolved. Other never will. Now back to mice. Researchers done some tricks, they pretend it is safe and beneficial, and mice even it feed by fat diat, still do not develop diabetes type 2, insulin resistance. Does anyone see the logic? I do.
      With every study group get some money to run research, right? It is easy to study mice then any human. How many mice died, no one count. How they recovered, and really did they recovered all? No one count of it. Mice easy to be replaced. What it was, product that blocked production of fatty acid, was it safe for mice? No one care about it. Finally, even if mice did not develop diabetes, did mice survived the studies? Did mice lived the rest of its normal life? No one study of it. No one has any interest in life of mice in lab cage.
Really, was diabetes resulted by high fat diet? What id diabetes resulted by high stress mice experienced in life in lab cage? No any answers to any one of this questions. No one really very interesting how long mice live after studies were completed. Next level of study is human studies and we became lab rats for Medical Complex Experiments. 
      Too much fat in the diet promotes insulin resistance by spurring chronic inflammation. But the researchers discovered, in mice, that when certain immune cells can't manufacture fat, the mice don't develop diabetes and inflammation, even when consuming a high-fat diet.
Well, take a look at the American NFL players. They eat very high nutrition, fat, protein, carbs. They need muscles, and they need energy. How many of them diabetics with insulin resistance? I do not know even one. There is diabetic NFL payer, but it is diabetes type 1, insulin dependent type of diabetes. Why? Because of plaer need energy to play ball. There is no energy if player treated with any medicine but insulin.So, where from the fact that diabetes type 2 resulted by high fat diet? No one evidence of it, only high scam and brain washing.
"The number of people with diabetes has quadrupled worldwide over the last 20 years," said senior investigator Clay F. Semenkovich, the Irene E. and Michael M. Karl Professor and director of the Division of Endocrinology, Metabolism & Lipid Research at the School of Medicine.
This is the FACT. The fact resulted by introducing type 2 diabetes back in 1980 by WHO. Before every diabetic was treated with insulin, regardless of any type of diabetes. Now Insulin is not the treatment regime for 90% of diabetic population. Of cause, Medical Complex do everything they can to keep situation under their control. Death rate of diabetics higher then any non diabetic population. And it is rising.
"An inhibitor of fatty acid synthase actually is now in clinical trials as a potential cancer treatment," he explained. "And other drugs have been developed to inhibit fatty acid synthase in diabetes, too. One possibility that our work suggests is that altering the lipid content in the cell membrane may help block cancer metastases and complications of diabetes."
They love two-in-one treatment. If treatment do not work for cancer they can say, it worked to treat diabetes type 2. If patient die because of cancer then they will day they prevented death from diabetes type 2 and its complications. In any way, they are those who run for payment, always. 
The researchers also plan to take a look at existing drug compounds that change the lipid composition in cells. Such medications failed in clinical trials, but they may have an effect on the membranes of macrophages and therefore may lower the risk of diabetes complications, Semenkovich said.
Do someone need more prove then they already show? As usual, read what they said with open mind, and ask questions.
 http://ift.tt/2eiTZts

Cause of inflammation in diabetes identified

Date:
November 2, 2016
Source:
Washington University in St. Louis
Summary:
Inflammation is one of the main reasons why people with diabetes experience heart attacks, strokes, kidney problems and other, related complications. Now, in a surprise finding, researchers have discovered, in mice, that when certain immune cells can’t manufacture fat, the mice don’t develop diabetes and inflammation, even when consuming a high-fat diet. 
 


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